Medial Humeral Condyle Fracture

Medial Humeral Condyle Fracture

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Injuries to the medial aspect of the distal humerus in young children can range from an avulsion fracture of the medial epicondyle to a much more serious Salter-Harris type IV fracture of the medial condyle. [1] Medial condyle fractures involve a fracture line that extends through and separates the medial metaphysis and epicondyle from the rest of the humerus; by definition, the fracture line must involve the trochlear articular surface. Medial condyle fractures must be distinguished from medial epicondyle fractures that involve the medial column but are extra-articular; these are separate entities.

The distinction between these two types of fractures is key to the selection of appropriate treatment. Both fracture patterns may be difficult to diagnose in young children, especially before the secondary ossification centers have formed. [2, 3, 4, 5, 6, 7, 8, 9, 10, 11, 12, 13, 14]  Inadequate reduction of the physeal growth plate and the joint surface cartilage in a medial condyle fracture can lead to serious complications.

The age of the patient, the extent of initial injury and displacement, and delay in initial treatment play important roles in the clinical outcome. For medial epicondyle fractures, nonoperative management of fractures displaced up to 15 mm does not appear to be associated with functional deficit unless there is entrapment of the epicondyle fragment in the joint. [15] Entrapment of the epicondyle fragment in the joint can block motion, and therefore, the fragment should be reduced.

For patient education resources, see the Breaks, Fractures, and Dislocations Center, as well as Broken Arm and Broken Elbow.

The humerus is a bone in the arm. The distal humeral physis (also called the growth plate) is located between the humeral metaphysis proximally and the epiphysis distally. The distal humeral epiphysis is bordered proximally by the physeal growth plate and distally by its articular surface with the ulna and radius. The humeral metaphysis is the growing portion of the humerus that lies between the epiphysis and diaphysis (the shaft or central part of a long bone). [16, 17]

The medial condyle of the humerus is the medial column of the distal expansion of the humerus that includes the following:

The trochlea is the distal medial articulating end of the humerus, which acts as a pulley for the ulnar trochlear notch to rotate around as the elbow is flexed. The coronoid fossa is the depression on the anterior surface of the medial condyle proximal to the trochlea that accommodates the coronoid process of the ulna. The olecranon fossa is the depression on the posterior surface of the medial condyle proximal to the trochlea, which accommodates the olecranon of the ulna.

The medial epicondyle is a prominent palpable process that projects medially from the trochlea and is the point of origin of the pronator teres and the common flexor tendon, which includes the flexor carpi radialis, the palmaris longus, the flexor carpi ulnaris, and the flexor digitorum superficialis. [18]  The medial epicondyle is the origin of the medial collateral ligamentous complex (see the image below). The medial supracondyle ridge is a bony ridge that runs proximally on the medial humerus from the medial epicondyle.

The capitellum, a rounded ball of bone adjoining the trochlea laterally, is the distal lateral articulating end of the humerus that articulates with the radial head. The lateral epicondyle is a prominent palpable process that projects laterally from the capitellum and is the point of origin of the common extensor tendon. The lateral supracondyle ridge is a bony ridge that runs proximally on the lateral humerus from the lateral epicondyle. The ulnar nerve, which runs in close proximity to the medial epicondyle, often may be palpated as a rounded cord just posterior to this bony prominence.

The elbow joint is a hinge-type synovial joint formed where the distal end of the humerus articulates with the proximal ends of the radius and ulna. It is a uniaxial joint with movements of flexion and extension. Normal range of motion is from 0° (full extension) to 135° (full flexion). Most functions of the elbow require motion of 30-130°. Consequently, a 30° extension lag has little functional significance. The normal physiologic carrying angle of the elbow in the anatomic position (full supination and extension) is approximately 165-170° (10-15° of valgus angulation).

Flexion is produced by the brachialis (the main flexor muscle) and the brachioradialis. In supination, the biceps brachii also flexes this joint; in pronation, the pronator teres assists in flexion. Flexion is limited by the apposition of the anterior surfaces of the forearm and arm, by tension of the posterior arm muscles, and by the collateral ligaments.

Extension is produced by the triceps brachii and is assisted by gravity and the anconeus . Extension is limited by impingement of the olecranon of the ulna on the olecranon fossa of the humerus and by tension of the anterior muscles and collateral ligaments.

At birth, a single cartilaginous cap covers the distal end of the humerus. During development, four separate ossification centers form at different times, as follows:

Further complicating this pattern of development, the trochlear ossification center is frequently composed of multiple irregularly sized foci that eventually coalesce into a single structure. The trochlear and capitellar ossification centers eventually fuse. The pattern of ossification at these multiple sites is highly variable.

In the developing elbow, fracture through the medial condyle involves part of the cartilaginous or partially ossified trochlea and the ossified medial epicondyle. With the insertion of the common flexor tendon of the forearm and the medial collateral ligament on the medial epicondyle, the fracture fragment tends to rotate around the axis of the epicondyle and can present at various degrees of rotation. Complete rotation puts the fracture surface facing the anterior and medial side of the elbow, with the medial epicondyle pulled distally and the articular surface facing posteriorly and laterally.

In addition, the surrounding soft tissues can be torn, and there may be damage to the articular surface of the ulna. Damage to the articular capsule or medial collateral ligament of the elbow may be present. The ulnar nerve and the vasculature surrounding the elbow joint are also at risk. The blood supply to the epiphysis enters with the attachment of the medial collateral ligament and the common flexor tendon. Separation of these structures at the time of injury or at surgery may lead to avascular necrosis.

The medial epicondyle fragment is usually displaced distally, though at least two cases of displacement of the fragment proximally have been reported. The fracture line usually involves only the apophysis; however, occasionally a fragment of metaphyseal bone is found attached to the avulsed fragment. If the fragment is incarcerated within the joint, the fragment may become adherent to the coronoid process of the ulna. When the fragment is incarcerated within the joint, the universal finding is a thick fascial band that binds the ulnar nerve to the underlying muscle. This thick fascial band is responsible for ulnar nerve dysfunction, either acutely or as a late finding.

Other elbow fractures may be associated with medial epicondyle fractures, and care must be taken to recognize the full extent of the injury. Associated injuries include radial neck fractures, olecranon fractures, and coronoid process fractures.

The following three possible mechanisms for medial condyle fracture [2, 3, 4, 6, 7, 8, 9, 10, 12, 19] have been described:

Three mechanisms of injury for medial epicondyle fractures [20, 21] have been proposed for an acute injury. All of them result in a partial or complete separation of the apophyseal fragment from the rest of the humerus.

The first mechanism of injury is a direct blow on the posterior medial aspect of the epicondyle that may be associated with fragmentation of the avulsed bone.

The second mechanism is a pure avulsion injury produced by the flexor muscles of the forearm (see the image below). This avulsion may occur in combination with a valgus stress that locks the elbow in extension. The classic example is the child that falls on the extended arm and hyperextends the wrist and fingers, placing more stress on the forearm flexors. The normal valgus angulation or carrying angle of the elbow tends to accentuate the forces responsible for the avulsion injury. This mechanism can also explain associated injuries, including a radial neck fracture with valgus angulation and greenstick fractures of the olecranon.

This second type of avulsion injury may be a pure muscular avulsion secondary to contraction of the forearm flexor musculature with an elbow flexed. This mechanism may be responsible for medial epicondyle fractures associated with arm wrestling and throwing a baseball.

The third mechanism proposed for medial epicondyle fracture is associated with a dislocation of the elbow (see the image below). In this mechanism, the ulnar collateral ligament provides an avulsion force that causes the medial epicondyle to fail.

Fracture of the medial condyle is very rare, especially when compared to frequency of other elbow fractures. In one study, radiographs of 589 elbow fractures in children younger than 16 years were reviewed. The most common fractures were the supracondyle fracture (55%), radial neck fracture (14%), lateral humeral condyle fracture (12%), medial epicondyle fracture (8%), and olecranon fracture (7%). [22] No cases of medial condyle fracture were reported. [23]

Fractures of the medial condyle are so rare that they receive little coverage in most popular textbooks and may not be mentioned at all in others. The incidence is described in the literature as being no higher than 1-2% of all elbow injuries in children. Most displaced medial condyle fractures occur when the trochlea is not ossified completely. Various studies have cited different average ages (ie, 10, 11, 9.5, and 7 years). Although a child of any age can sustain this fracture, it is most common during the developing years (ie, in children aged 7-14 years).

Medial epicondyle fractures are much more common than medial condyle fractures. In a large combined series of 5228 fractures of the distal humerus, medial epicondyle fractures constituted 14.1% of all distal humeral fractures and 11.5% of all fractures occurring around the elbow. Medial epicondyle fractures are four times as likely to occur in males, and most cases occur in children aged 9-14 years. Peak incidence is in children aged 11-12 years. The reported incidence of association with elbow dislocation reaches 55% in some series, and the fragment may be incarcerated in the joint in approximately 15-18% of cases. [24]

Children younger than 5 years tend to have nondisplaced fractures and good results. Older children tend to have more severely displaced fractures that involve the partially developed trochlear ossification center. Good results are obtained in patients who are seen early after injury and who have adequate reduction and fixation of their fractures. In patients seen late, proper reduction and immobilization are necessary to achieve satisfactory results; however, this is usually more difficult to achieve. Preserving full range of motion of the elbow depends on restoring the snug relation between the humerus and the ulna throughout the arc of motion.

Most children with medial epicondyle fractures treated either nonsurgically or surgically end up with good long-term functional results. Children with fracture displacement greater than 5 mm treated nonsurgically had results that were comparable to those of children who underwent surgical stabilization. Open reduction with internal fixation (ORIF) is required in children with medial epicondyle fractures and incarcerated fragments that block motion. Comminuted medial epicondyle fractures appear to do worse if the epicondyle is excised than if they are treated nonsurgically.

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John J Walsh, IV, MD Professor and Chairman, Department of Orthopedic Surgery, University of South Carolina School of Medicine

John J Walsh, IV, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, Christian Medical and Dental Associations, American Society for Surgery of the Hand

Disclosure: Nothing to disclose.

Joseph P Rectenwald, MD Orthopaedic Associates of Augusta, PA

Disclosure: Nothing to disclose.

Francisco Talavera, PharmD, PhD Adjunct Assistant Professor, University of Nebraska Medical Center College of Pharmacy; Editor-in-Chief, Medscape Drug Reference

Disclosure: Received salary from Medscape for employment. for: Medscape.

Murali Poduval, MBBS, MS, DNB Orthopaedic Surgeon, Senior Consultant, and Subject Matter Expert, Tata Consultancy Services, Mumbai, India

Murali Poduval, MBBS, MS, DNB is a member of the following medical societies: Association of Medical Consultants of Mumbai, Bombay Orthopedic Society, Indian Orthopedic Association, Indian Society of Hip and Knee Surgeons

Disclosure: Nothing to disclose.

Jegan Krishnan, MBBS, FRACS, PhD Professor, Chair, Department of Orthopedic Surgery, Flinders University of South Australia; Senior Clinical Director of Orthopedic Surgery, Repatriation General Hospital; Private Practice, Orthopaedics SA, Flinders Private Hospital

Jegan Krishnan, MBBS, FRACS, PhD is a member of the following medical societies: Australian Medical Association, Australian Orthopaedic Association, Royal Australasian College of Surgeons

Disclosure: Nothing to disclose.

Lee A Patterson, MD Orthopedic Surgeon, Carolina Bone and Joint Clinic, PA

Lee A Patterson, MD is a member of the following medical societies: American Medical Association and South Carolina Medical Association

Disclosure: Nothing to disclose.

Medial Humeral Condyle Fracture

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